When it comes to immunity, too much or too little is actually a disease.
The idea that a vaccine can be more effective than natural immunity is flawed at the outset. The immune system has multiple layers and strategies that cannot be fully utilized by an injectable vaccine. Training the mucosal immune system is virtually impossible with an injectable vaccine.
Crotty, Shane. "Hybrid immunity." Science 372.6549 (2021): 1392-1393.
These mucosal surfaces serve as the first line of defense against pathogens and are constantly exposed to a wide range of potential harmful agents, including bacteria, viruses, fungi, and parasites.
Mucosal immunity is distinct from systemic or peripheral immunity, which refers to immune responses occurring in the blood and lymphatic systems. The mucosal immune system has its own unique set of components and mechanisms to provide protection at mucosal surfaces. It plays a crucial role in preventing infections and maintaining immune homeostasis in the mucosal tissues.
Immunity is similar to blood clotting. It needs to be precise, not too much (leads to blood clots) or too little (excessive bleeding).
The video below with Jacinda Arden (Prime Minister - New Zealand) was taken from September 2022. It needs to be remembered that there was an extensive zero covid strategy taken by the government in New Zealand. This included limited movement in and out of the country and widespread vaccine mandates.
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Thank you. Question if I may: How does the super hybrid acquired immune tumult (SHAIT) disrupt / down regulate the activity of the innate immune system? Does any such interference at the innate immune level have anything to do with ADE, or is ADE almost all related to SHAIT’s effects at the acquired immune system level? Thank you
Pathogenic priming, known as Antibody Mediated Enhanced Disease or Antibody Dependent Enhancement (ADE)
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a body can produce antibody that when it later encounters the real germ, actually enhances disease.
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ADE occurs when the antibodies generated during an immune response recognise and bind to a pathogen, but they are unable to prevent infection. Instead, these antibodies act as ‘Trojan horse,’ allowing the pathogen to get into cells and exacerbate the immune response
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The autoimmune phenomenon would make the body’s own immune cells attack and kill other cells and tissues and organs. Thus patients would likely be a lot sicker with higher complications and death rates. More immunosuppressants would be required, leading to more secondary bacterial and fungal infections. Indeed, this clinical scenario is routinely seen in extremely sick and critical COVID patients.