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Comprehensive overview of COVID-19 research by Dr Philip McMillan, focusing on autoimmunity as the primary pathology in the disease.

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Are mild Covid Infections in the Vaccinated Necessarily good?

Prepare for the worst in Covid and hope for the best!

Dr Philip McMillan

Sep 17, 2023

The concern is regarding recurrent mild breakthrough COVID-19 infections across highly vaccinated regions. Could they indicate longer term risks?

The uptick in hospital admissions around the new Omicron variant is very concerning. Anticipating the outcomes, if negative, are very important.

Contribution of each spike-specific IgG subclass to the sum of all spike-specific IgG antibody levels. The percentages of each spike-specific IgG subclass relative to the sum of spike-specific IgG levels are shown for the vaccinated and COVID-19 cohorts. The numbers represent the mean percentages of individuals calculated by dividing each spike specific IgG levels with the sum of all spike specific IgG levels. Schematic representation of spike-specific IgG1%, IgG2%, IgG3% and IgG4% are shown in blue, orange, grey and yellow sectors, respectively. The size of the pie charts indicates the levels of all spike-specific IgG antibodies

Kiszel, Petra, et al. "Class switch towards spike protein-specific IgG4 antibodies after SARS-CoV-2 mRNA vaccination depends on prior infection history." Scientific Reports 13.1 (2023): 13166.

“Our results showed that EBV reactivation stimulates IgG4 production in a subset of Graves' disease, and in some cases, it may be the main source of IgG4. Therefore, high serum IgG4 levels and the infiltration of IgG4-positive plasma cells observed in IgG4-RD-like conditions may be present as a consequence of chronic inflammation and EBV reactivation.”

Nagata, Keiko, et al. "Epstein–Barr virus lytic reactivation induces IgG4 production by host B lymphocytes in Graves' disease patients and controls: a subset of Graves' disease is an IgG4-related disease-like condition." Viral immunology 31.8 (2018): 540-547.

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4 Comments

Lewis Coleman

Sep 18

The mammalian stress mechanism explains why the COVID symptoms appear in the aftermath of COVID “immunizations” and why this is a warning of more serious sequelae. The mRNA “immunizations” introduce what amounts to live coronavirus into systemic blood circulation, where the virus attacks the cells of the vascular endothelium and hijacks them to produce more virus. This is extremely dangerous, because normally the coronavirus is confined to the lung. When it invades systemic circulation and attacks the vascular endothelium, it causes the vascular endothelium to release immature fragments of von Willebrand Factor into systemic circulation, where it stabilizes and activates hepatic factor VIIIC to form “factor VIII” a gigantic protein complex that enzymatically interacts with factors VII, IX and X to accelerate thrombin generation to energize its conversion of factor X to factor XIII. Factor XIII then converts soluble fibrin into insoluble fibrin, a monomer that polymerizes into strands that entangle blood cells into clots. The virus also increases the permeability of the vascular endothelium, so that dangerous quantities of tissue factor in extravascular tissues escape into systemic circulation, where tissue factor stabilizes and activates hepatic enzyme factor VII, which enables and exaggerates the magnitude of the enzymeatic interaction of factors VII, VIII, IX and X. The combined activation of factors VII and VIII cause a “silent” increase in blood coagulability that can cause sudden onset of “disseminated intravascular coagulation” that often causes sudden death in young healthy people. www.stressmechanism.com

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Sfurl

Sep 17

Good explanation, can share this with a few people as they will grasp this better. Few of my older relatives struggle to understand a lot of the science and they need to get the basics so they can stay out of the shark pool. 😉

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