The silent Covid Vaccine death

Parallels to Hypertension, the silent killer

There is a lot of concern about harms of covid vaccines across the population and it implies that just ‘stopping the shots’ are the most important action.

Certainly correct that ongoing administration of covid vaccination needs to be stopped to reduce the risk of organ damage. Given that essentially the whole population will have either natural immunity or hybrid immunity, further vaccine booster administration may only increase risks.

Conversely, without going the next step of identifying the population at risk and already injured, this will only be a partial solution.

Many of the affected population will be unaware that there is a problem because there are often no symptoms.

Similar to hypertension, covid autoimmunity could be a silent killer.

Imagine that hypertension was previously ignored across the population. No longer having regular blood pressure checks and all hypertension medication stopped.

Within 5 to 10 years, the population outcomes for heart attacks, heart failure, strokes and aneurysms would be horrendous.

Historical Perspectives on Hypertension - American College of Cardiology

Roosevelt was documented as having hypertension at age 54, but did not receive treatment for another four years when he was prescribed phenobarbital and massage therapy for a blood pressure of 188/105 in 1941. In February 1945, Roosevelt had recorded blood pressures up to 260/150 at the time of the Yalta Conference, where he was noted to be in failing health with evidence of heart failure, including shortness of breath, orthopnea, lethargy and drowsiness. On the morning of April 12, 1945, a blood pressure of 300/190 was recorded after Roosevelt reported a severe occipital headache while sitting for a portrait session. He subsequently lost consciousness and died.

Read full article here >

In the context of covid vaccine silent organ damage, my research has pointed to low level autoimmunity around ACE-2 and a number of other proteins as the critical target.

The standout other protein is neuropilin-1.

“What conditions are associated with increased Neuropilin-1?”

There are several conditions in which neuropilin-1 (NRP1) levels may be increased. Some of these conditions include:

1. Cancer: NRP1 is known to be upregulated in various types of cancer, including pancreatic, prostate, and lung cancers. High levels of NRP1 expression have been associated with tumor progression, angiogenesis, and metastasis.

2. Inflammatory diseases: NRP1 has been implicated in the regulation of immune responses and inflammatory processes. Elevated levels of NRP1 expression have been observed in several chronic inflammatory diseases, including rheumatoid arthritis and psoriasis.

3. Cardiovascular disease: NRP1 has also been shown to play a role in cardiovascular disease. Increased levels of NRP1 expression have been observed in atherosclerotic lesions and in the circulation of patients with coronary artery disease.

4. Neurological disorders: NRP1 is also involved in neural development and function. Elevated levels of NRP1 expression have been observed in various neurological disorders, including Alzheimer's disease and Parkinson's disease.

Overall, the mechanisms underlying the upregulation of NRP1 in these conditions are complex and may involve various signaling pathways. Further research is needed to fully understand the role of NRP1 in these diseases and to develop targeted therapies that can modulate NRP1 expression or signaling for clinical benefit.

(Chat GPT response in italics)

This could manifest as persistent macrophage activation as reflected in elevated levels of ferritin, along with immune dysfunction.

Ferritin - Potential measure of a silent killer in the COVID-19 pandemic

The most critical scientific step is an acknowledgement of the issue with a determination to understand and find solutions urgently.

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